Dysentery is a potentially serious disease, caused by many different pathogens. There are different types of dysentery, including Amebic dysentery, Bacillary dysentery, Hemolytic uremic syndrome, and Entamoeba histolytica. It’s important to know the difference between each type so you can find the right treatment for your symptoms.
Bacillary dysentery
Bacillary dysentery is a severe gastrointestinal disease. It is a highly contagious disease, transmitted directly or indirectly through contaminated food. It can be fatal or lead to severe dehydration in children. Its symptoms include fever, abdominal pain, diarrhea, and vomiting. Vomiting is a particularly serious complication of bacterial dysentery as it can cause severe dehydration. Other symptoms of dehydration include dry mouth, sunken eyes, and pale skin. Children may be restless and have difficulty producing tears. They will also produce dark urine.
Treatment of bacillary dysentery begins with good hygiene. Wash your hands often and thoroughly. Avoid sharing personal items. Drinking sterile water is also essential. Boiling water or using chlorine tablets may be an effective way to achieve this goal. Using bottled water is also helpful in the prevention of bacterial infections. Also, stay away from people who are sick, and wash fresh fruits and vegetables thoroughly before eating them.
Weather variables may play a role in the transmission of bacillary dysentery. The impact of temperature and precipitation is, known to increase the risk of this bacterial infection. Researchers have previously studied the effects of weather conditions on the transmission of mosquito-borne diseases, such as dengue fever and malaria. In a recent study researchers, found that extreme precipitation levels were, associated with higher incidence of bacillary dysentery.
The incidence of bacillary dysentery was highest in northwest China and was lowest in southeast China and northeast China. The study area was defined by an arcgis map, which quantifies the association between daily meteorological factors and the incidence of bacillary dysentery in these regions.
Amebic dysentery
Amebic dysentery is an infection, caused by the Entamoeba histolytica parasite. It can be asymptomatic or cause a range of symptoms including fever, chills, diarrhoea, and abdominal pain. If the condition is severe, it can cause an abscess of the liver. It can also spread to the lungs. The disease can spread from one person to another via the faecal-oral route, diaper-changing, oral-anal sex, and infected water.
Symptoms of amebic dysentery may begin two to four weeks after exposure. Common symptoms include blood in the stools, abdominal pain, and nausea. In severe cases, a person may experience severe abdominal pain and bloody stools. Amebic dysentery can lead to a liver abscess. Although most cases do not require treatment, amebic dysentery is serious and requires immediate medical attention.
Amebic dysentery is, caused by a parasite in the intestine called Entamoeba histolytica. It is a highly contagious disease and is responsible for about 15 cases a year in Wisconsin. The symptoms of amebic dysentery may include diarrhea, bloody stools, abdominal pain, and fever. The infection is, usually treated with antibiotics.
People with symptoms of amebic dysentery are, advised to wash their hands thoroughly before eating, drinking water, and going to the toilet. If symptoms persist, the person is, advised to avoid oral-genital contact until the condition is fully under control. During an epidemic, the disease may spread more rapidly.
Hemolytic uremic syndrome
The United States Centers for Disease Control and Prevention (US CDC) estimates that non-O157 STEC strains are more common causes of diarrhea and hemolytic uremic syndrome in humans. In an effort to better understand these bacteria and their association with dysentery and hemolytic uremic syndrome, we reviewed the published literature for a comprehensive review of the rates of these infections.
Dysentery and hemolytic uremic Syndrome are caused by Escherichia coli strains producing shigatoxin. Shigatoxin-producing Escherichia coli is an emerging zoonotic pathogen with a range of symptoms. Although it is not known how these bacteria spread in the environment, they are associated with domestic animals and humans, and are responsible for a wide range of foodborne diseases.
Entamoeba histolytica
Amoebiasis is an infection caused by the parasite Entamoeba histolyticus, a type of one-celled organism. People can become infected by placing contaminated items into their mouths. This bacteria can cause cysts to grow in the intestines, which may lead to symptoms.
Diagnosis is often based on stool antigens and PCR, which can detect E. histolytica in the feces. Although these techniques are very sensitive and can differentiate between invasive and non-invasive infection, they can be expensive and lack standardization. Antigen detection may be done through stool antigen detection assays or serum immunoassays. In addition, immunofluorescence is another way to determine if the infection is E. histolytica.
People with AIDS and HIV are susceptible to developing invasive disease caused by E. histolytica. However, this infection is curable in such a population. Treatment options include medication and fluids. People who have diarrhoea after travelling should also be checked for amoebiasis.
In some cases, the parasite infects the gut wall and enters the bloodstream, where it can cause infection. In severe cases, the parasite may get into the liver and cause an abscess containing pus and dead liver tissue. Infected people may also experience fever, jaundiced skin, and abdominal pain.
This disease is, caused by the ingestion of infected feces. The infection usually begins with the ingestion of mature cysts or fecal contaminated food. Once inside the body, the parasite can cause infection in the small intestine.
Diagnosis and treatment for this condition depends on the type of E. histolytica you have. A specific diagnosis is important to limit the spread of the disease. Infected patients may be treated with luminal agents, such as paromycin, which can kill the parasites. The most commonly prescribed medications for invasive amoebiasis are nitroimidazoles. These drugs have longer half-lives than most antibiotics and are effective against trophozoites.
Shigella
The history of Shigella and dysentery covers both ancient and contemporary history. The story starts long before the first dysentery bacillus was, identified in 1898, and continues through the principal scientists who contributed to the understanding of the pathogenesis of this bacteria. Shigella has become an outstanding model for the study of invasive bacterial pathogens. Its evolution from a simple parasite into an invasive bacterium has been an invaluable resource in understanding the disease and developing new treatments.
Immune responses in children with Shigella-positive MSD were, associated with higher levels of cytokines, including IL-1b and IL-8. They were also found to be significantly higher than those in children without the disease. In addition, children with dysentery had increased levels of GM-CSF and IL-17A.
A pandemic of Shigella dysenteriae type 1 has occurred in four countries, notably Central America (1968-72), central Africa (the 1980s), and east Africa (the 1990s). These strains are resistant to multiple antibiotics, have high attack rates and case fatalities, and affect nearly all age groups.
While current WHO guidelines for children with dysentery seem to effectively manage this illness, they may be missing out on the opportunity to reduce the mortality and morbidity associated with Shigella infections in children. Further studies need to quantify the reduction in mortality associated with antibiotic therapy for children with non-dysentery Shigella infections.
Shigella and dysentery, caused by the same enteropathogen, Shigella flexneri. This bacterium infects the cells of the intestinal lining and invades them. The invading bacteria secret proteins, called invasins. These proteins are, involved in the triggering of cell death and in modulating the immune response of host cells.
